Answer: 64 codons
Explanation:
The triplet nature of the genetic code make for a possible 64 codons: 61 code for amino acids present in most polypeptide chains while the remaining 3 are special-sense codons, since they only signal for the termination of the polypeptide chain.
Thus, the pattern of the genetic code permits that a base appear more than once in a codon. For instance Adenine occurs twice in both AAU and AAC codons, which code for asparagine.
Explanation:
they are not transformed , and they die soon after they are still transfo4med because the nearby cells are transformed they change to a new cell with new characteristics. THEY ARE NOT TRANSFORMED AND THEIR CHARACTERISTICS ARE NIT ALTERED
<span>he Streptococcus pneumoniae capsule is vital for virulence and may inhibit complement activity and phagocytosis. However, there are only limited data on the mechanisms by which the capsule affects complement and the consequences for S. pneumoniae interactions with phagocytes. Using unencapsulated serotype 2 and 4 S. pneumoniae mutants, we have confirmed that the capsule has several effects on complement activity. The capsule impaired bacterial opsonization with C3b/iC3b by both the alternative and classical complement pathways and also inhibited conversion of C3b bound to the bacterial surface to iC3b. There was increased binding of the classical pathway mediators immunoglobulin G (IgG) and C-reactive protein (CRP) to unencapsulated S. pneumoniae, indicating that the capsule could inhibit classical pathway complement activity by masking antibody recognition of subcapsular antigens, as well as by inhibiting CRP binding. Cleavage of serum IgG by the enzyme IdeS reduced C3b/iC3b deposition on all of the strains, but there were still marked increases in C3b/iC3b deposition on unencapsulated TIGR4 and D39 strains compared to encapsulated strains, suggesting that the capsule inhibits both IgG-mediated and IgG-independent complement activity against S. pneumoniae. Unencapsulated strains were more susceptible to neutrophil phagocytosis after incubation in normal serum, normal serum treated with IdeS, complement-deficient serum, and complement-deficient serum treated with IdeS or in buffer alone, suggesting that the capsule inhibits phagocytosis mediated by FcÎł receptors, complement receptors, and nonopsonic receptors. Overall, these data show that the S. pneumoniae capsule affects multiple aspects of complement- and neutrophil-mediated immunity, resulting in a profound inhibition of opsonophagocytosis.</span>
Answer:
Option A, Glucose metabolism in the fetal brain increased
Explanation:
Usage of cocaine during pregnancy is prohibited. However, in case if cocaine is taken it disrupts the metabolism as it increases brain glucose metabolism to the level of an adult human being. Since glucose metabolism in brain is a sensitive process, any disruption in it can lead to unaccounted effects such as dysfunctional circulation, and pharmacological effect. Cocaine increases the dopamine by blocking the dopamine transporters thereby increasing the Vmax. Since the brain of a child has all kind of neurochemical components, cocaine intakes by a pregnant woman produces reinforcing effects in brain of fetus and also make it pharmacologically active
Hence, option A is correct
