Answer: Anconeus muscle
Explanation:
The anconeus is a small muscle found at the elbow. It is Attached to the humerus and ulna (it originates on the lateral epicondyle of the humerus and inserts on the olecranon process and superior portion of the ulna). Anconeus muscle allows the forearm to extend and it also provides support for the dorsal joint capsule and the ulna as well.
The radial nerve permits the anconeus muscle to carry all of those functions.
The group of plants that is most successful would probably be Angiosperms! They're also known as flowering plants or anthophytes.
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Answer:
1. The difference between the normal hemoglobin protein DNA sequence and the sickle cell hemoglobin DNA sequence is a base to base shift, in this case adenine (GAG) to thymine (GTG).
2. The difference affects the amino acid sequence of the protein by replacing glutamic acid (Glu) with valine (Val).
Explanation:
In sickle cell anemia, a change in the DNA nucleotide sequence is observed, where adenine is substituted by thymine, whose expression is the change in the amino acid sequence of globine β, incorporating valine instead of glutamic acid. This represents a molecular mutation - point mutation - by subtitution, which corresponds to missense mutation.
<u>Normal hemoglobin protein in a RBC</u>
DNA CTG ACT CCT GAG GAG AAG TCT
Amino acids Leu Thr Pro Glu Glu Lys Ser
<u>Sickle cell hemoglobin protein in a RBC</u>
DNA CTG ACT CCT <em>GTG</em> GAG AAG TCT
Amino acids Leu Thr Pro <em>Val</em> Glu Lys Ser
When GAG is transcribed to mRNA, the CUC codon is obtained, which codes for glutamic acid. Thymine substitution causes the DNA sequence to change to GTG, which is transcribed as CAC, the codon that encodes the amino acid valine. The <u>change from glutamic acid to valine in β-globin causes an altered hemoglobin, giving the abnormal erythrocytes observed in sickle cell disease</u>.
<span>It binds itself to the receptors present on the surface of the cell membrane, which activates the enzymes present inside cell. Then It passes through the cell membrane and attaches itself to a receptor protein present in the cytoplasm.
It passes on the signal to the secondary messengers inside the cell, which activate the receptors on the cell membrane.</span>
D; Hardy- Weinberg requires a very large population size so that genetic drift can be insignificant.
