Because when you put a part of the DNA and take it out, you can restrict an ability of that cell. When you cut out a piece of DNA from a pancreas beta cell that codes for insulin synthesis, you restrict the cells ability to make insulin, hence the name "restriction enzyme". I hope this is the answer that you are looking for and it comes to your help.
Answer:
Carbon can bond with many elements
Explanation:
Carbon can bond with many elements, including itself, to form macromolecules. Moreover, it can form four bonds, and that makes possible many different arrangements of the atoms in the molecules.
A is <em>wron</em>g. Carbon is among the seven smallest elements in the Periodic Table.
C is <em>wrong</em>. Carbon is found in living things because it can form macromolecules, not the other way around.
D is <em>wrong</em>. Carbon can bond with many other elements beside itself.
Using numerous tissue cultures will facilitate individuals perceive however cancer works, however it spreads, however it is attacked and eventually overwhelmed. Scientists will experiment with them while not being afraid that one thing can happen to the patient since they would not be victimization actual cancer patients, however rather a bit of tissue in a very research lab.
There are so many examples for that in different areas, like organic germanium compounds experiment carried out in our lab recently.
Here's one link: http://www.alfa-chemistry.com/products/organic-germanium-24.htm
Prader-Willi syndrome (PWS) is a gentic disorder which has an impact on numerous physiological systems. PWS affected individuals (specifically babies) experience delayed growth, significant hypotonia (low muscle tone), and feeding issues. It does effect circadian rhythms in mice models.
SNORD116, often referred to as HBII-85, is a non-coding RNA (ncRNA) molecule that contributes to the alteration of other small nuclear RNAs (snRNAs). Unlike the majority of other snoRNAs, SNORD116 is not significantly complementary to ribosomal RNA and is expressed widely in the brain (but not in PWS patients).
According to the studies, SNORD116 cause sleep defect in patients with Prader-Willi syndrome. Same observation was seen in mouse models too. Paternal expression of SNORD116 is thought to be a potential gene for the sleep disruptions/circadian rhythm’s that the majority of PWS sufferers.
To learn more about circadian rhythm click here
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