E. coli are a species of bacteria. Like all bacteria, they have a round piece of DNA that contains most of their genetic information, but also plasmids, that contain some more. Plasmid can be passed on from bacterium to bacterium and they contain information for fighting antibiotics. Tetracycline is such an antibiotic. The process of putting a plasmid inside a bacterium is called transformation. So what the above sentence really says, is that a bacterium just got a plasmid that is probably holding the genetic information to produce proteins that fight antibiotics like tetracycline. While regular strains of E. coli might not be able to deal with tetracycline, transformed strains will probably be able to survive it. Hence, the culture will have live bacteria and the bacteria that have a plasmid against tetracycline will be favored and survive.
What is your question, do you want to know what happens?
Organisms show dominant traits in most cases, hope this helps :)
Answer:
The correct answer will be option- Glycolysis.
Explanation:
Gluconeogenesis is a metabolic pathway which synthesizes glucose from non-sugar precursors. This pathway is activated in the low concentration of glucose due to less intake or completely absent to conserve energy.
This pathway is somewhat the reverse of the glycolysis when excess energy is present as a result of glycolysis, gluconeogenesis is inhibited and when no energy is present, gluconeogenesis is activated.
Thus, option- glycolysis is the correct answer.
Answer: Cystic Fibrosis
Explanation:
Cystic fibrosis (abbreviated CF) is an autosomal recessive genetic disease that mainly affects the lungs, and to a lesser extent the pancreas, liver and intestine, causing an abnormally thick, sticky mucus to build up in these areas. This mucus collects in the airways of the lungs and pancreas. The main cause of morbidity and mortality is pulmonary involvement, which accounts for 95% of deaths, mainly due to repeated infections caused by bronchial obstruction due to the secretion of very thick mucus.
This build up of mucus causes life-threatening lung infections and serious digestive problems. It is one of the most common types of chronic lung disease in children and young adults, and is a life-threatening disorder; patients often die from lung infections due to <em>Pseudomonas</em> or <em>Staphylococcus</em>.
<u>It is a hereditary disease produced by a mutation in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR) protein. This protein is involved in the passage of chlorine ion through cell membranes and its deficiency alters the production of sweat, gastric juices and mucus. </u>The disease develops when neither allele is functional. Over 1500 mutations have been described for this disease, most of which are small deletions or point mutations; less than 1% are due to mutations in the promoter or chromosomal rearrangements. However, many people carry the CF gene, but do not have any symptoms. This is because a person with this disease must inherit 2 defective genes, 1 from each parent.
<u>There is no curative treatment, however there are treatments that allow the improvement of symptoms and extend life expectancy. In severe cases, the worsening of the disease may necessitate a lung transplant.</u>
