Growth delays
When giving methylphenidate to nursing patients, monitoring appetite suppression or growth delays is important. Methylphenidate is a member of the stimulant class of medications. They function by raising brain activity, particularly in regions that aid with attention and behavior regulation.
In order to treat children with attention deficit hyperactivity disorder, methylphenidate is employed (ADHD). They can focus better and it aids with hyperactivity and impulsive conduct. Additionally, people with ADHD or narcolepsy are treated with it (a sleep disorder).
Only prescriptions are accepted for methylphenidate. It is available as early release or prolonged release pills and capsules. To assist their ADHD, some kids might require both prolonged and rapid release methylphenidate.
Here's another question with an answer similar to this about attention deficit hyperactivity disorder (adhd): brainly.com/question/4099245
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<em><u>Answer:</u></em>
Malaria is a life-threatening disease. its tightly transmitted through the bite of an infected anopheles mosquito. When this mosquito bites you the parasite is released into your bloodstream.
When the parasite is inside your body they travel to the Liver, they mature and after several days they affect your red blood cells.
<em><u>SYMPTOMS OF MALARIA.</u></em>
a) Chills
b) High fever
c) Headache,e.t.c.<em><u /></em>
Answer:
Correct answer is c. It is the final electron acceptor in the aerobic respiration.
Explanation:
Oxygen is a substrate of the aerobic respiration, but it is not the only one. Glucose is also a substrate.
Oxygen is used in the cells to be the final electron acceptor, this means that receives the electrons from NADH and FADH2. That is why, when there is no oxygen available for aerobic respiration, the NADH and FADH2 cannot be oxidized and therefore remain in their reduced form. As a consequence, they cannot be re-utilized during different cellular processes that are NAD+ and FAD dependant, such as glycolysis, pyruvate oxidation and cellular respiration. This means that the ATP synthesis stops.
Oxygen itself does not transport any electrones, this are transported by the cytochrome complex in the mitochondrial membrane. But oxygen is key in receiving those electrones, therefore a very important piece of the electron transport across the mitochondria.
Answer:
<u>Ethanol prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u>
Explanation:
Ethylene glycol is an organic compound usually used in antifreeze solutions, solvents and cleaners.
It is abused during self-destruction and accidental intakes.
<u>In the body, ethylene glycol is acted upon by alcohol dehydrogenase and is converted into glycolate and oxalate.</u>
Glycolate and oxalate are both nephrotoxic/ kidney damaging substances. Oxalate precipitates calcium oxalate stones in the kidney. Ethylene glycol poisoning also causes high anion gap metabolic acidosis.
In order to prevent ethylene glycol poisoning, the patient is infused with ethanol, ethanol <u>prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u> In this way, ethylene glycol is not metabolized and the formation of nephrotoxic substances is prevented. Alcohol dehydrogenase instead reacts with ethanol to form acetaldehyde.
