Question: Is the pre-synaptic or the post-synaptic neuron damaged? Explain your rationale in detail (in other words, detail how you came to your conclusion). You're going to have to really think on this one. It's not something you can look up in the text. You will need to use what you have learned about the autonomic nervous system and some critical thinking to arrive at the answer.
Horner's Syndrome riddle: Ach is the neurotransmitter at all ANS preganglionic fibers, including SNS. The pt has decreased sweating and pupil constriction, indicating blockage of SNS transmission. When the MD gave ACh it induced sweating, but not pupil dilation. This is because the sweat glands are an exception in normal SNS transmission, as the postganglionic fibers utilize ACh as their neurotransmitters. Other SNS pathways utilize NE as their neurotransmitter. So ACh acted directly on the sweat gland to produce sweating, but could not stimulate the typical postsynaptic neurons to release NE. When the MD gave NE he saw pupil dilation, but not sweating, indicating the NE worked directly at the iris to produce pupil dilation. This indicaates the problem was with the postsganglionic neuron. If the preganglionic neuron were the problem injecting ACh would have solved both problems.
Answer:
<em>The number of chromosomes is maintained through the process of mitosis during asexual reproduction. </em>
Mitosis can be described as a process in which a cell divides and produces two daughter cells which are exactly similar to the parent cell. The chromosome number of the daughter cells will be the same as the parent cells. No crossing over or recombination occurs during this type of cell division. Hence, mitosis is an effective way to maintain the chromosome number of organisms which reproduce asexually.
RNA brings genetic code to the ribosomes to direct the synthesis of the amino acid sequence.
2 times pro year and sometimes 3 times
