Answer:
The bugs in the soil or on the plants will die
Explanation:
The bugs and some of the plant will be infected some will die some will stay alive it all depends on how strong the pesticide is and consummation of the crop will sometimes result in a dangerous turn of event to your stomach if not washed properly.
I would say D. Osteoarthritis, presbyopia
The diagnosis will be variant creutzfeldt jakob disease or as called as vCJD. The variant creutzfeldt jakob disease is an intermittent and fatal human neurodegenerative illness. The ingestion of food of bovine origin contaminated with the agent of bovine spongiform encephalopathy which is a disease of cattle and has been strongly connected to the existence of vCJD in humans.
Answer:
The answer is 20 degrees then the temperature will start going to other organs
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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