It's genetic variation just took the quiz
Answer:
12:3:1
Explanation:
<em>The typical F2 ratio in cases of dominant epistasis is 12:3:1.</em>
<u>The epistasis is a form of gene interaction in which an allele in one locus interacts with and modifies the effects of alleles in another locus</u>. There are different types of epistasis depending on the type of alleles that are interacting. These include:
- Dominant/simple epistasis: Here, a dominant allele on one locus suppresses the expression of both alleles on another locus irrespective of whether they are dominant or recessive. Instead of the Mendelian dihybrid F2 ratio of 9:3:3:1, what is obtained is 12:3:1. Examples of this type of gene interaction are found in seed coat color in barley, skin color in mice, etc.
- Other types of epistasis include <em>recessive epistasis (9:3:4), dominant inhibitory epistasis (13:3), duplicate recessive epistasis (9:7), duplicate dominant epistasis (15:1), and polymeric gene interaction (9:6:1).</em>
This would directly affect the epidermis because blisters will develop within the surface of the skin. Having blisters on the epidermis will decrease the function of the skin to be an effective barrier to disease. This is because blisters are open wounds that are prone to infection. As infection passes through these openings, it will affect the dermis and the other layers of the skin. Bullae is actually an auto-immune skin disease.
The name is in the answer: Flood Stage
Hope this helps!
