Answer:
by providing healthy nutritional source and put them in pure culture
Answer:
Es probable que a través del tiempo la población de piojos también se vuelva resistente al segundo pijojicida. Los piojos resistentes se generan por un proceso de selección natural
Explanation:
La evolución es el proceso mediante el cual los organismos cambian (evolucionan) con el tiempo con el fin de adaptarse a sus condiciones ambientales. La selección natural opera seleccionando aquellos individuos mejor adaptados al ambiente en el cual cada individuo se desarrolla. Los individuos adaptativamente superiores poseen más chances de sobrevivir y de reproducirse, trasmitiendo de este modo sus genes a la descendencia. Las mutaciones representan la fuente principal de variabilidad genética en la forma de nuevos alelos y genes. Las mutaciones que son beneficiosas y confieren una ventaja adaptativa son seleccionadas por selección natural. En este caso, la resistencia al primer piojicida se debió a la aparición de una o más mutaciones las cuales confirieron resistencia al primer compuesto. Con el tiempo, la frecuencia de los individuos portadores de mutaciones beneficiosas en la población de piojos fue incrementándose paulatinamente (como resultado del proceso de selección natural). Es muy probable que este proceso evolutivo también se repita con el segundo piojicida.
Sudoriferous Glands are under the control of the sympathetic nervous system and are located in the dermis.
Sudoriferous Glands are also called as sweat glands which are of two types Eccrine or Apocrine. These two reside within the dermis and consists of secretory cells and central lumen into which material is secreted.
The Sympathetic Nervous system is one of the three division of the Automatic Nervous System arises near the middle of the spinal cord in the intermediate nucleus .
Sudoriferous Gland are small tubular structures that is helpful in producing sweat. These are exocrine gland and produces sweat on epithelial surface through duct.
Eccrine are present everywhere except ear canal, prepuce, labia minora and smaller than apocrine and excrete directly onto the surface of the skin and its proportion decreases with the age.
Apocrine are found in the armpit, areola in the ear and the eyelids. the secretory portion is larger than the eccrine glands and secrete swear into the pilary canal of the hair follicle.
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Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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