Answer:
The correct answer is - epiphyseal plate.
Explanation:
Achondroplastic dwarfism is genetic disorder that occurs Due to the failing in elongation of the long bones of the limbs. This failing of the elongation takes place due to the reduced hyperplasia and hypertrophy of cartilage in the epiphyseal plate, that present at the end of long bones and produce the bone.
The epiphyseal plate is the site of growth of the bones for longitudinal growth by the hypertrophy, and proliferation of cell. Change or error in the gene cause the reduce hypertrophy and proliferation of the cell of cartilage in the epiphyseal plate that leads to the Achondroplastic dwarfism.
Thus, the correct answer is - epiphyseal plate.
<span>1. Movement 2. Flexibility 3. Protection 4. Support</span>
The correct answer is D: I and II only.
Viruses have protein capsids, which protect their genetic material. This capsid sometimes is covered by viral envelopes which have glycoproteins on their surface. Glycoproteins help in the process of binding to the host cell and infecting it. In this example, virus III has the structure of a bacteriophage and it does not seem to have glycoproteins. Bacteriophages use their tail fibers to attach to the bacterial host and inject their genetic material. On the contrary, viruses I and II have glycoproteins sticking out of their envelopes.
Specific monoclonal antibodies are produced by hybridoma cells, which form when B lymphocytes and myeloma cells combine.
<h3>How are hybridoma cells produced and give rise to monoclonal antibodies?</h3>
Hybridoma or phage-display technology can be used to make monoclonal antibodies. In order to elicit an immunological response, a mouse is first immunized using a specific antigen in hybridoma technique. In hypoxanthine-aminopterin-thymidine (HAT) media, B cells from the spleen of the immunized mice are isolated and united with myeloma cells that have undergone immortalization. Myeloma cells are specifically chosen to be HAT-sensitive and non-antibody secreting. Hybridomas, the resulting fusion product, secrete antibodies and are immortal.
Unfused B cells also perish since they are not immortal and cannot survive in the HAT mixture with unfused myeloma cells. Then, these hybridomas are diluted into single cell wells and put through an antibody screening process. These cloned B cells generate particular monoclonal antibodies.
Learn more about hybridoma cells here:
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