Answer:
1. Acetylcholine binds to receptors on the motor end plate
2. Ligand-gated channels open leading to depolarization
3. End plate potential triggers an action potential
4. Transverse tubules convey action potentials into the interior of the muscle fiber
5. Calcium is released from the sarcoplasmic reticulum
6. Calcium ions bind to troponin, which then moves tropomyosin
Explanation:
Acetylcholine (ACh) is a signaling molecule (neurotransmitter) that binds to receptors on muscle cells. This binding triggers the opening of ligand-gated sodium channels, thereby ions enter into muscle cells, which causes the depolarization of the sarcolemma and thus promotes the release of Ca2+ ions from the sarcoplasmic reticulum. The myoneural junction, also known as the motor endplate, is the site of synaptic contact between a motor axon and a skeletal muscle fiber. The endplate potential is the voltage that produces the depolarization of muscle fibers when ACh molecules bind to their receptors in the cell membrane. This depolarization spreads in the sarcolemma through transverse tubules (T tubules) and thus generates an action potential. Finally, this action potential induces the release of Ca2+ in the sarcoplasmic reticulum, which activates troponin protein and induces muscle contraction.
Answer:
area of ocean or lake surface over which the wind blows in an essentially constant direction, thus generating waves the term also is used as a synonym for fetch length which is the horizontal distance over which wave-generating winds blow
Explanation:
Answer:
e. infection causes lymphocytes to divide more rapidly
Explanation:
The cell cycle includes interphase and M phase which in turn together produce daughter cells from the existing parent cells. DNA replication occurs during the S phase of interphase to ensure that the daughter cells obtain the identical DNA present in the parent cell.
Lymphocytes are one of the types of white blood cells and are involved in cell-mediated and antibody-mediated immune responses. These cells are present in the blood as well as in lymphatic tissues. During the given experiment, lymphocytes exhibited a higher rate of incorporation of labeled nucleotide after the introduction of a pathogen in the culture. This suggests that the introduction of pathogen triggered the cell division in lymphocytes to produce more lymphocytes to fight the infection.
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Explanation:
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