Answer:
a. True, b. False, c.True, d. True
Explanation:
a. Base excision repair is started by a DNA glycosylase that recognizes the changes and removes the altered base by cleavage of the glycosidic bond binding the base and the deoxyribose sugar together.
b. Nucleotide excision repair works by a cut-and patch mechanism that removes their heavy lesions, including pyrimidine dimers and nucleotides . Endonucleases are responsible for the lesion of the damaged strand.
c. Nucleotide excision repair is initiated by the proteins namely UvrA, UvrC, and UvrB in Escherichia coli.
-UvrD (helicase II) later removes the damaged strand
-DNA polymerase I (PolI) fills in the resulting gap.
d. DNA glycolases removes the damaged nitrogenous base.
-It leaves the sugar-phosphate backbone intact and thus creating an apurinic/apyrimidinic site, which is commonly referred to as an AP site.
e. Xeroderma pigmentosum complementation group A(XPA)
-This is an essential protein in the nucleotide excision repair pathway.
- It helps to make a pre-incision complex along with other proteins.
I think it’s the first one
The answer is (b). I think
Answer: It can detect chemical cues produced by the predator Notonecta glauca.
Explanation:
The answer to your question is,
if the oxygen poor blood mixes back with oxygen rich blood, that means that oxygen poor blood is not going back to the lungs to swap CO2 (carbon dioxide) with O2 (oxygen), and this swapping is vital to tissue survival, since oxygen is what keeps your tissues, muscles, and organs alive. so if this blood mixed, ultimately your organs are not receiving enough oxygen and will become starved of it, this can kill you and lots of people every year get heart surgery because their atrium or ventricles in their heart rupture
-Mabel <3
