Answer: due to insufficient exocytosis in the type II alveolar cells
Knockout of the circadian clock protein per1 exacerbates hypertension and increases kidney injury in dahl salt-sensitive rats resulting in worsening of the SS hypertensive phenotype (1).
A circadian clock factor BMAL1, significantly found in kidneys, is known to affect blood pressure. Moreover, a diet high in salt is associated with immune cell infiltration in the kidney and consequently increases blood pressure. The BMAL1 factor is also responsible for expressing various genes. Mice, when treated with BMAL1 showed aging phenotypes like muscle weakness, changed gait, decreased mobility, inactivity, etc.
PER1 is a circadian clock transcription factor and is regulated by aldosterone. Nice on PER1 knock-out develops reduced sodium excretion with day and night differences. This is done in response to a high-salt diet.
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Answer:
The correct answer is - Control microcosms did not contain living moss, while experimental microcosms did contain living moss.
Explanation:
The difference between the control microcosms and experiment Microcosms is, the presence of the living moss in the experimental group whereas the control group does not contain living moss.
The independent variable in this research setting is the presence or absence of the living moss and for the control group, the mosses are filtered out and only added the water.
<span>The size of plasmids<span> ranges
between 1 and 200 kbp (kilo-base pairs) and a DNA fragment that can be
inserted into a </span>plasmid vector is up
to 20 kbp. Artificially constructed plasmids are often used as vectors in
genetic engineering to clone and amplify (or express) particular genes. </span>
