Answer:
A sea breeze or onshore breeze is any wind that blows from a large body of water toward or onto a landmass; it develops due to differences in air pressure created by the differing heat capacities of water and dry land. As such, sea breezes are more localised than prevailing winds. Because land absorbs solar radiation far more quickly than water, a sea breeze is a common occurrence along coasts after sunrise. By contrast, a land breeze or offshore breeze is the reverse effect: dry land also cools more quickly than water and, after sunset, a sea breeze dissipates and the wind instead flows from the land towards the sea. Sea breezes and land breezes are both important factors in coastal regions' prevailing winds.[1] The term offshore wind may refer to any wind over open water.
Wind farms are often situated near a coast to take advantage of the normal daily fluctuations of wind speed resulting from sea or land breezes. While many onshore wind farms and offshore wind farms do not rely on these winds, a nearshore wind farm is a type of offshore wind farm located on shallow coastal waters to take advantage of both sea and land breezes. (For practical reasons, other offshore wind farms are situated further out to sea and rely on prevailing winds rather than sea breezes.)
Explanation:
Answer:
c. Sarcomere
Explanation:
Sarcomere is one of the basic components of striated muscle that allows muscle contraction. That is, it is the contractile unit in the muscle cell.
The sarcomere is made up of a protein complex, the most important of which are actin and myosin. In the presence of Ca2 +, the ends of the myosin bind to nearby actin molecules and fold with great velocity. The actin filament then moves to the center of the sarcomere, triggering the approach of the two Z lines. This causes the sarcomere to shrink and, to a large extent, lead to contraction of the entire muscle.
Answer:
Fructose 2,6‑bisphosphate (F26BP) activates phosphofructokinase‑1 (PFK -1) and inhibits fructose 1,6‑bisphosphatase (FBPase)
Explanation:
Fructose 2,6‑bisphosphate (F26BP) is a metabolite that is produced with an increase in glucose, hence increasing the availability of fructose-6-phosphate. With, the increased concentration of F26BP, it increases the affinity of PFK- 1 to fructose-6-phosphate, thereby activating glycolysis which enhances the catabolism of glucose. In contrast, F26BP inhibits the activity of fructose 1,6‑bisphosphatase (FBPase), hence inhibiting gluconeogenesis. Gluconeogenesis (formation of glucose) will not be need since there is the presence of glucose in the system.
In summary, fructose 2,6‑bisphosphate (F26BP) reciprocally controls the enzymatic activity of phosphofructokinase‑1 (PFK -1) and fructose 1,6‑bisphosphatase (FBPase); it inhibits gluconeogenesis by inhibiting the enzyme, FBPase and activates glycolysis by activating the enzyme PFk -1
