Fertilization of the egg cell by the sperm usually takes place in the fallopian tube. The fertilized egg then travels to the uterus and implants in the endometrium.
<h3>What are fallopian tubes?</h3>
- Fallopian tubes are also called oviducts or uterine tubes. It is the passage through which the egg enters the uterine cavity from the ovary.
- Fallopian tubes are part of the reproductive tract. They have a smooth muscle wall, an inner mucous membrane, and an outer layer of loose supporting tissue (serosa).
<h3>Why does fertilization take place in the fallopian tubes?</h3>
The fallopian tube (oviduct) regulates fertilization through sperm induction and sperm hyperactivity. Sperm induction is achieved by rheotaxis, thermotaxis, and chemotaxis. Rheotaxis is caused by tubal fluid that creates a current flow from the tubal ampulla to the tubal isthmus.
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Desmosomes
Desmosomes are cell junction that acts as anchors and distributes tension through a cellular sheet and reduces the chance of tearing when it is subjected to great mechanical stress.
Desmosomes are cell structure by which two adjacent cells are joined. Desmosomes are specialized for cell to cell contact and strong adhesion. The inactiveness of desmosomes can lead to diseases of the skin and heart. Desmosomes are found in tissue that undergo high mechanical stress, such as bladder tissue, epithelial, and cardiac muscle tissue.
Answer:
C
Explanation:
its temperature because it's the best answer
Answer:
A rainwater containing carbon dioxide dissolves underground rock
Explanation:
Water in an underground rock is groundwater. Underground water can also erode and deposit material. Rainwater absorbs carbon dioxide (CO2) as it falls down to the surface. The CO2 groundwater creates landforms by getting rid of rock.
Answer:
Abstract
Down syndrome (DS) is associated with aberrations in genetic, morphological, biochemical and physiological characteristics. A number of genes located on human chromosome 21 (HSA21) encode proteins which are thought to be involved in numerous metabolic pathways, e.g., phosphofructokinase, cystathionine β-synthase etc. Perturbations of the metabolic pathways may lead to altered drug metabolism in DS individuals. We present a review of metabolic aberrations linked to HSA21 genes in DS. We particularly focus on drug disposition, efficacy, sensitivity and toxicity of anti-leukaemic agents including methotrexate, glucocorticoids, anthracyclines and cytarabine in DS leukaemia. The different outcomes of therapy due to differential drug response, varied drug toxicity and treatment related mortality in DS leukaemia is a subject of much research and speculation. Altered drug response in DS individuals may stem from differences in pharmacokinetics, pharmacodynamics and pharmacogenetics. Further large-cohort studies in different age groups dissecting metabolic and molecular pathways involved in drug response may increase our understanding in this regard and stipulate pharmacotherapies in DS.
