Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
Answer:
wild animals collectively; the native fauna (and sometimes flora) of a region.
Explanation:
I hope this helped :)
D surface tension because, adhesion is sticking, b makes no sense for this question, and heat has nothing to do with it either, because it distributes it's weight throughout its legs so it doesn't sink (it doesn't weigh much so this works.)
Hope this helps don't forget to hit that heart :)
Answer:
conformational change in troponin
Explanation:
The troponin complex plays an essential role in the thin filament regulation of striated muscle contraction.
Answer/Explanation:
Cells of a similar type are organized together to make tissues. Different types of tisues come together to perform a similar function as part of organs.
For example, take the organ the stomach. There are various tissues that make up the stomach, including, for example, mucosa, and muscle tissue.
The mucosa tissue of the stomach is made up of secretory cells such as parietal cells, which secrete hydrochloric acid, and foveolar cells which produce mucus that protects the lining of the stomach.
