He demonstrated that SCD and sickle cell trait were due to the presence of abnormal 8-globin polypeptides in red blood cells. He demonstrated that the electrophhoretic mobility of B-globin from patients with SCD was different from that of healthy individuals. He demonstrated that both parents of multiple patients with SCD had low levels of sickled red blood cells. He hypothesized that SCD was a recessive trait and that the parents of patients with SCD would be heterozygous carriers. He demonstrated that the difference between B-globin polypeptides in individuals who were healthy and those with SCD is an amino acid substitution. He performed a peptide fingerprint analysis on B-globin from individuals with 84 84 and 89 88, which identified the segment of B-globin that was changed by the BS mutation. James Neel Linus Pauling Vernon Ingram
hope it helps..
Answer:
either of two masses of gray matter lying between the cerebral hemispheres on either side of the third ventricle, relaying sensory information and acting as a center for pain perception.
Explanation:
Answer:
A. Outer Membrane B. Peptidoglycan C. Cytoplasmic Membrane
Explanation:
I can't really give an explanation for this one but if you look up gram negative bacteria and go to images you can find a photo that is very similar to the one you show that has labels.
Bacteria are decomposers when they are turning the dead material in decaying organisms into nutrients. They become producers when they provide those nutrients for plants.
Answer:
Jordan
Explanation:
In most cases, an O parent and an AB parent will have only A or B kids.