An emergency room nurse is speaking with a patient who is exhibiting symptoms of an ischemic stroke that started two hours ago. The client mentions having had a cholecystectomy six weeks prior and using labetalol, digoxin, and warfarin. What element puts the patient's thrombolytic therapy at risk?
What is ischemic stroke?
An ischemic stroke is the loss of brain tissue (cerebral infarction) brought on by insufficient oxygen and blood flow to the brain as a result of an arterial blockage. An artery leading to the brain can get blocked, frequently by a blood clot or a fatty buildup brought on by atherosclerosis, leading to an ischemic stroke.
In patients with ischemic stroke, thrombolytic therapy must be started within three hours. If the patient underwent surgery within the last 14 days, she is not qualified for thrombolytic therapy. Labetalol and digoxin do not preclude thrombolytic treatment.
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Answer:
B. Benzodiazepines will decrease heart rate, blood pressure, and muscle tension.
Explanation:
I calculated it logically
Answer:
Explanation:
he sports physical therapist (SPT) is uniquely qualified to participate in the provision of preparticipation physical examinations (PPE). The PPE is recommended prior to athletic participation and required by many jurisdictions. There is little research to support the process and components; however, a number of professional organizations have recommendations that direct the PPE process.
Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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