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Andre45 [30]
2 years ago
5

How is a trophic cascade (top down regulation) different from the traditional trophic pyramid (bottom up regulation)?

Biology
1 answer:
Sidana [21]2 years ago
5 0

Top-down effects imply control through predation, including fishery, while

bottom-up effects imply control through food abundance, often thought to be driven by climate or nutrient load.

<h3>What is Top down regulation ?</h3>

The top-down effect refers to a higher trophic level influences the community structure of a lower trophic level through predation.

<h3>What is Bottom up regulation ?</h3>

The bottom-up effect means that a lower trophic level in the biological network affects the community structure of higher trophic levels by means of resource restriction

Learn more about Top down and bottom up regulation here:

brainly.com/question/18996262

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n200080 [17]

Answer:

Biology examines the structure, function, growth, origin, evolution, and distribution of living things. It classifies and describes organisms, their functions, how species come into existence, and the interactions they have with each other and with the natural environment.

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3 years ago
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How would the plants in a specific environment be impacted if there was a sudden drop in the amount of bacteria present in that
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How does oxygen production relate to the rate of photosynthesis?
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How do fungi benefit from being able to reproduce both asexually and sexually?
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4 0
3 years ago
If you treated embryos with a Notch inhibitor how would this affect the specification of venous and arterial endothelial cells?
Alisiya [41]

Answer:

d. all endothelial cells would be venous

Explanation:

The Notch signaling pathway is a cell signaling system which consist basically of 4 NOTCH genes ( NOTCH1, NOTCH2, NOTCH3, and NOTCH4) each of them having the information to build a particular cell membrane receptor protein.

It has been demonstrated in zebrafish embryos that Notch signaling are highly determinant factors during vascular development, and a key function of these genes was specifically to regulate the differentiation of arterial fate in endothelial cells.

Notch signaling-deficient embryos showed a loss of expression of arterial markers such as ephrinB2 from arterial vessels with an accompanying expansion of venous markers into normally arterial domains. In contrast, embryos in which Notch signaling had been ectopically activated, meaning an activation out of "proper" place (where not expected, e.g. venous endothelial), exhibited the contrary phenotype, this is a suppression of vein-specific markers with ectopic expression of arterial markers in venous vessels.

To summarize, when Notch is supressed (in this case inhibited with a specific Notch inhibitor) arterial fate of endothelium is not followed, and venous fate is stablished. Thus, endothelial cells (those expected to form venous tissues but also arterial ones) would specified as venous.  

4 0
3 years ago
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