Answer:
This is the diffusion (loss of water vapor) from the stomata (most cases) of a plant to its external environment.
It involves movement of water vapor down the water potential gradients; that is ; from higher water potential inside the leaf to lower water potential in the surrounding environments.
its cools the plant’s internal temperature as water vapour conveys heat out of plants' cells.
Answer:
A heterotroph is an organism that doesn't make their own food.
Explanation:
Answer: -20
Explanation:
Here is the equation to solve:
(-107)-(-97)+(-10)
First and foremost, we should note that:
- × - = +
- × + = -
Therefore, (-107)-(-97)+(-10)
= -107 + 97 - 10
= 97 - 107 - 10
= 97 - 117
= -20
Answer: a. Increasing the surface area for diffusion
Explanation:
When the air is inhaled through the nostrils, the air containing the oxygen the air diffuses inside the alveoli. The alveoli are the tiny sacs where air enters and exchange of gases takes place. From the alveoli the oxygen enters into the blood stream. The oxygen is utilized by the cells for respiration. The increase in network and number of the alveoli will increase the surface area for the diffusion of oxygen and hence, will speed up the respiration process.
The regulatory protein in muscle responsible for binding to calcium for contraction to proceed is troponin.
Troponin is a crucial player in the regulation of muscle contraction by calcium: The actin-tropomyosin-troponin complex, which makes up the thin filaments of striated muscles, is the only structure that can bind calcium. Actin filaments are also connected to troponin, a calcium-binding protein that controls contraction in skeletal or cardiac muscles.
Three parts or subunits of troponin:
- Tropomyosin-binding subunit (Tn-T)
- Inhibitory subunit (Tn-I)
- calcium-binding subunit are the (Tn-C)
NMR spectroscopy was used to determine the atomic structure of the troponin head.Troponin and tropomyosin are both necessary for the control of calcium in striated muscles. Troponin or tropomyosin mutations result in the well-known cardiac myopathy.
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