Answer: B
Explanation:
The question wants to know what the expression is directly dependent on.
The sequence of genetic expression goes like this:
DNA -> RNA -> protein
If we want to know the root cause of expression, we must look at the DNA, not the RNA or protein, because DNA determines the RNA and protein expression.
The Shape of subunits in DNA -> Sequence of bases coded for by the ribosome with RNA -> Arrangement of amino acids in the protein synthesized.
Why not C: Number of chromosomes present isn't a factor, because most humans, barring those with a genetic disorder, have 23.
Answer:
Change in chromatin structure-------Acetylation of histones facilitates transcription
Activity of transcription apparatus------Transcriptional activators increase promoter activity by interacting with the RNAPII holoenzyme
RNA processing---------Alternative splicing produces different transcripts from the same gene.
RNA interference-------A siRNA and the transcript it targets is transcribed from the same gene.
Initiation of translation-------Limited availability of initiation factors prevents translation of mRNA.
Chicken nuggets and fries
Answer:
The best answer to the question: If every gene has a tissue-specific and signal-dependent transcription pattern, how can such a small number of transcriptional regulatory proteins generate a much larger set of transcriptional patterns? Would be:
Because transcriptional regulators, which are the ones responsible for initiating, and stopping, transcription of RNA into protein, often work in pairs, one goes with the other, and thus increase the regulatory capabilities over gene expression so that the genes translated into RNA and then transcribed into aminoacids in protein chains, actually code for the correct protein types.
These regulators will both stand, as appropriate, on a specific gene to promote its transcription, or prevent it, depending on the different signaling mechanisms received.
