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kupik [55]
3 years ago
11

Why does prolonged exposure to UV light from the sun or tanning beds increase the risk for getting skin cancer?

Biology
2 answers:
LenKa [72]3 years ago
8 0

Answer:

Option B, increased exposure to UV light increases the risk of a mutation occurring

Explanation:

Ultraviolet radiations (UV) coming from the sun are radioactive waves that affect the skin. They not only cause skin infection, irritation, but are are also potential source of cancer causing agent . Some times these radiations on prolonged exposure  causes genetic mutations in the skin cells which continues though ages.

Ultraviolet rays are short wavelength radiations and the shorter is the wavelength of any radiation, the greater is the potential harm caused by it.

Hence, UV rays are harmful.

Option B is correct

Lilit [14]3 years ago
7 0
B. Increased exposure to UV light increases the risk of mutation occurring.
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Which best describes the offspring of protists?
Hunter-Best [27]

Options found elsewhere:

-always genetically identical to one parent

-always genetically identical to both parents

-never genetically identical to one parent

-sometimes genetically different than both parents

Answer:

-sometimes genetically different than both parents

Explanation:

Protists are eukaryotes that cannot be classed as animals, plants, or fungi. They are quite diverse. Examples include amoeba, plasmodium, and slime mould.

Because they are so diverse, protists also have very different modes of reproduction. Some protists undergo asexual reproduction, where they simply make a copy of themselves without a mate. Others undergo sexual reproduction.

Therefore, the only option that can be correct is "-sometimes genetically different than both parents" .

If they were always genetically identical to one parent or both parents, that would mean that sexual reproduction could not be possible. If they were never genetically identical to one parent, that would mean that asexual reproduction could not be taking place.

7 0
3 years ago
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How can we fix man made structures from affecting our ecosystem ? any ideas!!!???​
ale4655 [162]

It can affect our ecosystem in many ways, think about the animals. Where are they going to live? If trees, get demolished to create a hotel, or a gas station.

3 0
3 years ago
Please help its due in a hour!!!!!
garri49 [273]

Answer:

1. The difference between the normal hemoglobin protein DNA sequence and the sickle cell hemoglobin DNA sequence is a base to base shift, in this case adenine (GAG) to thymine (GTG).

2. The difference affects the amino acid sequence of the protein by replacing glutamic acid (Glu) with valine (Val).

Explanation:

In sickle cell anemia, a change in the DNA nucleotide sequence is observed, where adenine is substituted by thymine, whose expression is the change in the amino acid sequence of globine β, incorporating valine instead of glutamic acid. This represents a molecular mutation - point mutation - by subtitution, which corresponds to missense mutation.

<u>Normal hemoglobin protein in a RBC</u>

DNA                 CTG ACT CCT GAG GAG AAG TCT

Amino acids     Leu  Thr   Pro   Glu   Glu   Lys   Ser

<u>Sickle cell hemoglobin protein in a RBC</u>

DNA                 CTG ACT CCT <em>GTG</em> GAG AAG TCT

Amino acids     Leu  Thr   Pro   <em>Val</em>   Glu   Lys   Ser

When GAG is transcribed to mRNA, the CUC codon is obtained, which codes for glutamic acid. Thymine substitution causes the DNA sequence to change to GTG, which is transcribed as CAC, the codon that encodes the amino acid valine. The <u>change from glutamic acid to valine in β-globin causes an altered hemoglobin, giving the abnormal erythrocytes observed in sickle cell disease</u>.

6 0
2 years ago
Answers amoeba sisters: video recap meiosis
Schach [20]

1. Interphase is an important and the longest phase of the cell cycle during which the cell prepares for division by coping its DNA. It is metabolic phase of the cell, in which the cell grows, obtains nutrients and metabolizes them. There are three stages of interphase: G1 (the cell growth), S (replication of DNA, chromosomes are copied) and G2 (preparation for division). Without this phase, genetic material wouldn’t be ready for the process of meiosis  and haploid gametes couldn’t be created.

2. Homologous chromosomes are the similar but not totally identical chromosome pairs that an organism receives from its two parents. During the prophase I of meiosis they pair up: each chromosome aligns with its homologue partner via link-chiasmata ( the two match up at corresponding positions). Those homologue pairs separate during a first stage of cell division (meiosis I-reduction of chromosomes number, from diploid to haploid), while sister chromatids separate during a second stage (meiosis II).

3. Crossing over is a process in which homologous chromosomes trade their parts. Crossing over is process of genetic recombination where DNA is cut and then repaired. Cut and repair of homologous chromosomes allow them to exchange some of their genetic information. As a consequence of crossing over, new arrangement of maternal and paternal alleles on the same chromosome is achieved. It is the way to create varations.

4. During the metaphase I, homologue pairs are lined up comparing to metaphase II where individual chromosomes are lined up. It is because during the meiosis I homologue pairs separate and chromosome number reduce from diploid to haploid. On the other hand, during the meiosis II, sister chromatids separate.

5. Nondisjunction is the consequence of cell division, where there is no properly separation. There are  different forms of nondisjunction:  

• failure of a pair of homologous chromosomes to separate in meiosis I,  

• failure of sister chromatids to separate during meiosis II.

After nondisjunction, resulting daughter cells are with abnormal chromosome numbers -aneuploidy.


4 0
3 years ago
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HELLLPPP PLEASE I NEED TO FREAKING PASS​
Yuliya22 [10]

Answer:

I would say everything's correct except the second one, I believe that one is false :) I hope you do well and sorry if i'm wrong!

Explanation:

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