Answer:
Lysosomes.
Explanation:
HEXA (Hexosaminidase A) is the functional enzyme and encoded by the HEXA gene. The main function of this enzyme is the hydroxylation of the gangliosides.
The mutation in this gene leads to the lysosomal storage disease. The GM2 starts accumulating in the lysosomes and causes Tay Sach's disease. This is a genetic disorder that leads to the breakdown of the cells of the spinal cord and the brain.
Thus, the answer is lysosomes.
Answer:
Potential & Kinetic energy.
Explanation:
The water used gains potential energy overtime before it is converted to mechanical (or kinetic energy). This can be explained by the water having increased potential energy as it flows at an equal altitude, but converting to kinetic energy as it flows downhill, or over the dam.
This kinetic (or mechanical) energy is then converted to traditional electricity. This process is also an example of a renewable resource being utilized for everyday things (such as powering cars, homes. or charging batteries).
Answer:
Neurilemma
Explanation:
Schwann cells are the neuroglia that produces myelin sheaths. There is a formation of multiple layers of the glial plasma membrane around axons. The cytoplasm and nucleus of Schwann cells from the outermost layer while the inner part has multiple layers of the Schwann cell membrane. The outer nucleated cytoplasmic layer of the Schwann cell that encloses the myelin sheath is called neurolemma. It is found only around axons in the PNS. Its function is to serve in the regeneration of injured axons by the formation of a regeneration tube.
I think it's hydrothermal vents? hope its right because I have short term memory loss.
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
