The secondary succession is triggered by a natural event (forest fires for example) that destroys an established ecosystem. The destroyed forest will then undergo a regrowth process. This secondary growth may look bizarrely different from the way how the original forest used to look like. There are several stages involved after the forest os being burned. The first stage involves the colonization of massively burned places by plants and fungi. The following stage starts when minuscule plants such as grasses dwell in the forest. And then, huge plants that require lots of direct sunlight will move in. Lastly, new trees will continuously grow up to a point where they makeup a forest that remains in time except there are major interventions.
Answer:
Transitional fossils show how a particular taxa accumulated adaptations to fit particular environments and/or ecological niches
Explanation:
Transitional fossils are fossilized remains of taxonomic groups/species that illustrate an evolutionary transition between a known version of a taxa/species and the current taxa/species. Transitional fossils are fundamental because they can be clearly differentiated from the ancestral group as well as of its derived descendant group. For example, there exist transitional fossils known as "mammal-like reptiles"(i.e., therapsids that gave rise to the true mammals), which are clearly different from current mammals.
The metabolism of glucose involves the breakdown of glucose molecules to form the needed energy by the cell called ATP.The process follows the order of;
1.Glycolysis
2.Citric acid cycle
3.Oxidatize phosphorylation.
Answer:
Leptin is a protein hormone composed of 167 amino acids of the cytosine family, it is synthesized mainly by white adipose tissue. The leptin receptor is related to that of receptors for cytosines (since it uses Janus or JAK type kinases and STAT-3 proteins as intracellular mediators of transcription pathway), it is predominantly found in the hypothalamus (particularly in the arcuate nucleus and to a lesser extent in the nuclei dorsomedial and ventromedial), hippocampus and cerebellum. There is evidence that leptin acts at the level of the arcuate nucleus, preventing the formation of NPY.
Explanation:
Leptin is produced exclusively in adipose tissue cells in a wide variety of species, including humans, and its concentration is higher in overweight than in lean individuals. The biological actions of leptin can be classified into two groups, those that are exerted in the tissues of the central nervous system, mainly the hypothalamus, and those that are carried out on the peripheral tissues. The former regulate body weight downward, decrease food intake, increase basal energy expenditure and modify some neuroendocrine functions such as reproduction, while the latter have effects on proliferation, differentiation and metabolism of peripheral tissues. The leptin that reaches the hypothalamus, in addition, inhibits protein synthesis and the secretion of neurons producing NPY / AgRP of the arcuate nucleus and stimulates the synthesis and secretion of those containing POMC. NPY (neuropeptide Y) is produced in the hypothalamic arcuate nucleus. This neurotransmitter arrives through the projections of the neurons of said nucleus to the paraventricular nucleus, which is the area where it is released. There are several studies that have shown that an increase in NPY leads to an increase in the sensation of hunger and thus to hyperphagia and obesity. Leptin acts at the level of the arcuate nucleus, preventing the formation of NPY. The leptin receptor lacks enzymatic activity in its intracellular domain. Instead, it is linked to members of the janus kinase family, which belong to a class of tyrosine kinases. The binding of the ligand activates the Jak kinase and leads to the phosphorylation of certain cytoplasmic proteins. Within these proteins there is a class of cytoplasmic transcription factors called signal transducers and activators of transcription (STAT).
