Penicillins disrupts bacterial cell wall synthesis.
<h3>
How does penicillin affect bacterial cell walls?</h3>
- Penicillin kills bacteria by inhibiting the proteins which cross-link peptidoglycans in the cell wall .
- When a bacterium divides in the presence of penicillin, it cannot fill in the “holes” left in its cell wall.
- β-Lactam antibiotics, including penicillins, cephalosporins, monobactams, and carbapenems, are distinguished by a lactam ring in their molecular structure and act by inhibiting the synthesis of the peptidoglycan layer of bacterial cell walls.
- Penicillins work by bursting the cell wall of bacteria. Drugs in the penicillin class work by indirectly bursting bacterial cell walls.
- They do this by acting directly on peptidoglycans, which play an essential structural role in bacterial cells.
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Answer:
The cell is the smallest structural and functional unit of living organisms, which can exist on its own.
Explanation:
Answer - <span>C) Trees and shrubs provide shade, which keeps the water temperature cooler. Lower temperatures allow the water to hold more oxygen, which in turn creates a healthier habitat for the trout.</span>
Reasoning - The back story is if the water temperature is being changed. It will affect the trout indirectly warming the water. In turn if you have shades and shrubs to provide it for cooler areas it will preserve O2 a Certain Habitat for the Species.
Answer:
True
Explanation:
A mutation is any alteration in the genetic sequence of the genome of a particular organism. Mutations in the germline (i.e., gametes) can pass to the next generation, thereby these mutations can increase their frequency in the population if they are beneficial or 'adaptive' for the organism in the environment in which the organism lives (in this case, an insect/bug). The mutation rate can be defined as the probability of mutations in a single gene/<em>locus</em>/organism over time. Mutation rates are highly variable and they depend on the organism/cell that suffers the mutation (e.g., prokaryotic cells are more prone to suffer mutations compared to eukaryotic cells), type of mutations (e.g., point mutations, fragment deletions, etc), type of genetic sequence (e.g., mitochondrial DNA sequences are more prone to suffer mutations compared to nuclear DNA), type of cell (multicellular organisms), stage of development, etc. Thus, the mutation rate is the frequency by which a genetic sequence changes from the wild-type to a 'mutant' variant, which is often indicated as the number of mutations <em>per</em> round of replication, <em>per</em> gamete, <em>per</em> cell division, etc. In a single gene sequence, the mutation rate can be estimated as the number of <em>de novo</em> mutations per nucleotide <em>per</em> generation. For example, in humans, the mutation rate ranges from 10⁻⁴ to 10⁻⁶ <em>per </em>gene <em>per</em> generation.
