to leave their wavelengths unchanged
They are neither, they do not have the mechanism to ingest or produce food.
Answer:
The correct genotype of the two pure lines and the F1 is:
A⁺A⁺B⁰B⁰C⁰C⁰D⁰D⁰ and A⁰A⁰B⁺B⁺C⁺C⁺D⁺D⁺
The number of additive alleles on each genotype are two and six respectively.
Explanation:
Locus( plural form . loci) are fixed point on a chromosome in which genes are located. These genes are specific genetic material or genotype.
Now;
If we decide to designate the allele of the four loci into either additive (⁺) or non-additive(⁰); we have the following :
Let's the allele of the four loci to be
A⁺/A⁰, B⁺/B⁰, C⁺/C⁰ and D⁺/D⁰
However, from the diagram below; we deduce that the correct genotype for the two pure lines and the F1 is as follows:
A⁺A⁺B⁰B⁰C⁰C⁰D⁰D⁰ and A⁰A⁰B⁺B⁺C⁺C⁺D⁺D⁺ and the number of additive alleles on each genotype are two and six respectively.
The cross between both F1 traits will yield an heterozygous individual for the offspring. i.e A⁺A⁰B⁺B⁰C⁺C⁰D⁺D⁰ with only four additive allele
Answer:
The experiment will be faulty
Explanation:
To set up a control group, you pick individuals at random( so you don't just get a group to be of the same character, or opinion).
The control group is vital in every experiment because, sometimes our hypothesis could be mere coincidence or assumptions. So the control group will be compared to the test group to see if the results are correct. Plus look at it this way, without a control group, your hypothesis wouldn't be a theory.
Answer:
Mutations that activate the kinase portion of the receptor result in a receptor that is constantly phosphorylated. This causes constitutive activation of downstream signaling and the resulting cell growth and proliferation
Explanation:
The epidermal growth factor receptor (EGF receptor) is a type of receptor tyrosine kinase that recognizes and binds different ligands (including the Epidermal growth factor), which triggers its dimerization through the interaction of the extracellular domains. In humans, several mutations in genes encoding receptor tyrosine kinases have been associated with cancers. It is for that reason that mutated receptor tyrosine kinase genes are well-known oncogenes. Moreover, mutations in several receptor tyrosine kinase genes that lead to constitutive activation by phosphorylation have also been identified. In this case, it is expected that a mutation in the EGF receptor leads to constitutive phosphorylation of the mutant protein, constitutively activating downstream signaling.