Answer:
It allows flexibility (adjustment) and survival of a population in the face of changing environmental circumstances.
Explanation:
Answer:
I Think this answer will help
Explanation:
Soda ash/sodium hydroxide injection
This treatment method is used if water is acidic (low pH). Soda ash (sodium carbonate) and sodium hydroxide raise the pH of water to near neutral when injected into a water system. Unlike neutralizing filters, they do not cause hardness problems in treated water.
Answer:
sarcoplasmic reticulum deteriorates and ATP production is stopped
Explanation:
Rigor mortis is the third stage of death characterized by stiffening of joints and muscles in body. The stiffening occurs because muscles are not able to return to the relaxed state. There are two reasons for rigor mortis, depletion of ATP and increase in calcium concentration in cytosol. Due to these factors the actin-myosin crossbridge is not able to break and the muscles remain in contracted state.
Sarcoplasmic reticulum deteriorates and calcium is released into the cytosol. Sarcolemma ( covering of muscle fiber ) also breaks down releasing extra calcium into the cytosol. Calcium is responsible for formation of actin-myosin cross bridge and when its concentration increases the bridge is formed continuously leading to stiffening of muscles and joints.
Greetings!
Option A would be correct. The most likely outcome, (if) ribosomes stopped functioning properly, would be Cell growth stopping, or slowing down.
~Lauv
Answer:
Cancer cells achieve proliferative immortality by activating or upregulating the normally silent human TERT gene (hTERT) that encodes telomerase, a protein with reverse transcriptase activity that complexes with other proteins and a functional RNA (encoded by hTR, also called hTERC) to make a ribonucleoprotein enzyme.
Explanation:
A rare cell that escapes crisis almost universally does so by reactivating telomerase and this cell can now become a cancer cell with limitless potential to divide. Almost all cancer cells have short telomeres and thus inhibitors of telomerase should drive such cancer cells into apoptotic cell death. Yet, each time a cell divides, the telomeres get shorter. When they get too short, the cell no longer can divide and becomes inactive or "senescent" or dies. This process is associated with aging, cancer, and a higher risk of death.
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