Answer:
epididymis
ductus deferens
ampulla of ductus deferens
seminal gland
ejaculatory duct
urethra
Explanation:
Epididymis is a tube that connects the back side of the testicles to the vas deferens (ductus deferens). The vas deferens is the connecting link between epididymis and urinary tract/urethra and is responsible for transporting sperm to the urethra. Seminal vesicles are small glands that lie above the prostate gland and make up more than 50% of the total semen. Seminal vesicles are connected to the vas deferens through ejaculatory duct. This duct empties sperm and semen into the urethra
Hence, the correct order is
epididymis
ductus deferens
ampulla of ductus deferens
seminal gland
ejaculatory duct
urethra
Answer: The death of frogs would send a ripple through the ecosystem. The number of animals that eat the frogs will die off, then the animals that eat them will go hungry, and their populations will be devastated as well. This would just keep going up the food chain
Explanation:
Invasive bullfrogs upset the balance of predator-prey interactions and competition for food and shelter, but they also may be worsening another ecological disaster in the amphibian world. ... The invasive American Bullfrog can act as a vector, harboring this fungus, with no ill effects, and spread it to other amphibians
hopefully this helps :)
Consumers An organism that gets energy by eating other organisms. All animals are consumers. They must take in food because they cannot make their own. Decomposer an organism that gets energy by feeding on dead organisms and wasters.
The null hypothesis here is "the insertion of the wild type MSH2 gene does NOT reduce the number of <em>C. neoformans </em>C3 and C6 cells". This hypothesis is not supported by Figure 2, thereby the alternative hypothesis is accepted.
In the scientific method, a null hypothesis is a plausible explanation that states that there is no statistically significant difference between a certain feature and/or variable of a particular population.
In this case, the null hypothesis indicates that there are no differences associated with the insertion of the wild-type MSH2 gene variant (i.e., the normal allele) in the growth rate of <em>C. neoformans</em> strains on a medium containing a toxic chemical (mutagenic agent).
Figure 2 does not support the null hypothesis because the growth of wild-type MSH2 gene inserted <em>C. neoformans</em> C3 and C6 strains is inhibited when they grow on a medium containing a toxic chemical, thereby this hypothesis is rejected and the alternative hypothesis is accepted.
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