The correct option is (d)They are each made of matter. Some are compounds made of one type of element. Some are pure atoms.
Any substance with mass and volume is considered matter because it occupies space. Water is a substance that is created when Hydrogen and Oxygen are combined in a fixed ratio (2 H: O) with a chemical link. The elemental metal silver can be found in nature in its metallic form. Carbon is an element, and carbon dioxide as a compound is created when elements of carbon and oxygen are joined in a specific ratio to form the gas.
They are each made of matter. Some are compounds made of one type of element. Some are pure atoms.
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Answer:
The correct answer is - Control microcosms did not contain living moss, while experimental microcosms did contain living moss.
Explanation:
The difference between the control microcosms and experiment Microcosms is, the presence of the living moss in the experimental group whereas the control group does not contain living moss.
The independent variable in this research setting is the presence or absence of the living moss and for the control group, the mosses are filtered out and only added the water.
Answer:
The membrane is actually semi-permeable.
Explanation:
The membrane being semi-permeable means that, not all the things come in and out of the cell, only if it's really needed. The cell works in a gradient way, and that means that, to balance things, the cell will put in it a substance in a very high concentration outside of it, just to balance things. And this would happen to potassium, the cell would put it inside to balance this gradient, and once all sides have the same amount of potassium, it will stop, and the gradient is finally equal.
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.