Answer and explanation;
-The insulin receptor itself has decreased kinase activity leading to fewer binding sites for singling effectors to become activated.
Therefore; the signaling effectors are less likely to be activated.
-Additionally,, feedback inhibition decreases signaling effector binding to receptor.
-The lack of activation of these initial signaling effectors prevents any subsequent steps from happening.
-We can thus conclude this by saying that higher insulin concentrations are necessary to obtain the same level of signaling leading to physiological effects that would exist in someone without type-2 diabetes.
-Also the requirement for higher insulin concentration means that the cells that respond to insulin are resistant to its effect.
Since diabetics are not able to make enough insulin, scientists use gene engineering to make it for them. they insert the gene that codes for the production of insulin in the plasmid (circular DNA of bacteria ) to create recombinent DNA, and the bacteria will asexually reproduce, making more and more of the insulin in a short amount of time! hope this helped it took me a long time to understand the whole process in this unit!
My answer is the Vacoules, but I might be wrong.
Answer:
Cell membrane of prokaryotes and Inner mitochondria of eukaryotes
Explanation:
Electron transport chain is a part of oxidative phosphorylation which is the third step of aerobic cellular respiration in living organisms. The ETC is a complex of proteins and mobile electron carriers. It occurs when electron is being transferred from one electron carrier to another, starting from NADH and FADH2. A proton pump which is used to synthesize ATP is generated at the end.
This ETC process occurs in the cell membrane of prokaryotic cells due to the fact that they lack a membrane-bound Mitochondrion. ETC occurs in the inner membrane of the mitochondria called CRISTAE in eukaryotic cells.
<span>Statistically, which adolescent is most apt to experience menarche first? The </span> Answer Is ( C)
