Answer:
The most likely explanation for the truncated polypeptide -due to the substitution of cytosine for adenine- is that mutation introduced a stop codon in the middle of the gene.
Explanation:
A codon consists of three nucleotides -in the RNA chain- whose order determines a specific amino acid. Not all codons code for amino acids, as there are termination codons, also called stop codons, which are UAG, UGA and UAA.
If in Manny's computer model, the substitution of cytosine by adenine produced a termination codon, the synthesis of a peptide is stopped prematurely resulting in a truncated peptide.
Learn more:
Stop codon brainly.com/question/6183177
A biologist .....they study living things only.
Answer:
The structure of one or more proteins produced by the organism.
Explanation:
Genetic mutation can also can cancer 5 - 10% of the time.
* More than 40 proteins and glycoproteins involved in the complement system are synthesized by the liver, macrophages, epithelial cells, they are present in the blood in plasmatic form, membrane, some have an enzymatic activity, regulator or membrane receptorThese are elements of the humoral innate immune response, they fight infections, purify immune complexes and apoptotic bodies.
<span>There are indeed three ways to activate the complement:</span>
Classical pathway: Activated by Immunoglobulins in immune complexes, aggregated Immunoglobulins, DNA, CRP, apoptotic bodies .......it involves nine fractions, starting with C1, then C4, C2, C3, to form a classical C5 convertase, then, activation of C5, C6, C7, C8, C9.
Alternative pathway: activated by polysaccharides (bacterial endotoxin), vascular wall poor in sialic acid, aggregated IgE ...C3b like is the first component in the alternate channel cascade, it will create an amplification loop, and form an alternative C5 convertase.
Lecithin pathway: Activated by mannose, fucose (carbohydrate of microorganisms)The first component is the complex MBL / MASP1 / MASP2: "mannose-binding protein": works according to the same principle as the complex C1 of the classical way (MASP2 cleaves the C4 and the rest of the cascade is equivalent to that of the classical way).
the three ways have the same outcome: A C5 convertase (formed by one of the pathways) cleaves C5 into C5a and C5b: C5b is deposited far from other fractions on the antigenic surface. The fixation of C5b in the cell is followed by that of C6, C7, C8, and C9 (9 molecules of C9): formation of the membrane attack complex (MAC) ==> Death of the cell by osmotic shock
Explanation:
Silent mutations occur when the change of a single DNA nucleotide within a protein-coding portion of a gene does not affect the sequence of amino acids that make up the gene's protein.
