Answer:
- Calcium binds to troponin C
- Troponin T moves tropomyosin and unblocks the binding sites
- Myosin heads join to the actin forming cross-bridges
- ATP turns into ADP and inorganic phosphate and releases energy
- The energy is used to impulse myofilaments slide producing a power stroke
- ADP is released and a new ATP joins the myosin heads and breaks the bindings to the actin filament
- ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, starting a new cycle
- Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Explanation:
In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Answer: To be produced by meiosis
Explanation:
Answer:
1977 is when people first knew about archaebacteria
Answer:D.D.) Suspend the cells in liquid agar culture and monitor which culture continues to grow.
Explanation
Options A-C are correct except D.
→Increase in Telomere length is indicative of abnormal growth, cancer cells can reactive telomerase to increase cell's length.
→Once a monolayer is reached; this a stage of a complete normal cell division , a cell that goes beyond this stage must be mutated. assuming all other factors are constant .
Uncontrolled cell division is an indication of virulent growth in mutated cells.
<u>Cancer cell requires a ,medium of living host to thrive, alga medium will not support growth for carcinoma cells -the correct answer. </u>
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Completed Que.
:Imagine that you are a graduate student working in a cancer lab. You accidentally mix unlabeled tubes of carcinoma cells with tubes of normal epithelial cells. Which of the following is NOT a possible strategy to allow you to distinguish which tubes contain carcinoma cells?
A.)Monitor telomere length over time during multiple cell divisions.
B.) Plate the cells in culture and look for the culture that stops dividing once a monolayer is reached.
C.) Monitor the rate of cell division.
D.) Suspend the cells in liquid agar culture and monitor which culture continues to grow.
Answer:
Procedural memory
Explanation:
Isabel has to recall the information about using a manual stick shift. For this reason, she is using long-term, explicit, declarative and episodic memory system.
Human memory system can be broadly classified as: sensory memory, short-term memory and long-term memory.
Long-term memory is used to recall information. If the information are about facts, events and experiences then explicit, declarative and episodic memory helps to recall all information. This will help Isabel to recall all information about driving a car with a manual stick shift and she will be able to drive her sister's car.
(Answer taken from brainly.com/question/11839849)
