Answer: pathogen–host coevolution
Explanation:
A major driver of evolution is Reciprocal coevolution between host and pathogen. Rather than pathogen, one-sided adaptation to a nonchanging host, high virulence specifically favoured during pathogen–host coevolution. In all of the independent replicate populations under coevolution, the pathogen ( B. thuringiensis ) genotype BT-679 with known nematocidal toxin genes of C. elegans and high virulence specifically swept to fixation but only some of them go under one-sided adaptation,
so relative change in B. thuringiensis virulence was greater than the relative change in C. elegans resistance is due to the elevated copy numbers of the plasmid containing the nematocidal toxin genes
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Antagonsitic effect/interaction/response
In order to combat antiobiotic resistance, and to possibly enhance the activity of antibiotics, they are sometimes used in combinations during treatment. However, three possible responses or effects can manifest.
First is antibiotic synergy, where the combined effect of the antibiotics enhances the activity/potency of the treatment compared to when the antibiotics are administered singly.
The effect is also distinguished from another type of response, which is additive effect, where the combined effect of the antibiotics is more or less equal to the combined activity/potency of each of the antibiotic when applied singly. Antibiotic synergy results in even greater enhancement of the activity of the combined antibiotics compared to additive effect.
Lastly, there is the antagonistic effect or response, where the combined effect of the antibiotics results in the weakening of the potencies of the antibiotics relative to the combined (additive effect) potencies of each of the antibiotics.
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❖ This statement is false.
PROKARYOTE cells are cells that don't contain a nucleus, whereas a EUKARYOTE has cells that have a nucleus.
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It gets transferred through out animals
hope this helps
in my knowledge biotic factors could affect this process of photosynthesis
