The cause is a tick-borne disease,
According to the symptoms, its certainly a Lyme disease.
The bullseye (also called erythema migrans) around a tick bite leads to a bacterial infection: <span><em>Borrelia burgdorferi</em>.
Concerning the tick, it's from the genus <em>Ixodes.</em></span>
The correct answer is Karl will get sick and Jose will not this time, but neither will get this influenza again since they both will have acquired active immunity to it.
In the case of Jose artificially active immunity has been induced due to vaccination. A vaccine instigates a primary response against the antigen without resulting in any signs of the disease. On the other hand, Karl will acquire active immunity naturally in future, as when an individual get exposed to a live pathogen, he or she develops the disease, and turns immune as an outcome of the primary immune response.
Hello there.
Question: <span>Where are proteins digested and absorbed?
Answer: It is the small intestine.
Hope This Helps You!
Good Luck Studying ^-^</span>
The only statement that is not true concerning the definition of life in general is that "They are stable over time." This can be proven in the early years of earth's life wherein different microorganism and macroorganism lives. Over time, they evolved into another species that is most suitable to the kind of envionment the earth is evolving.
TAND-1 and TAND-2 of TAF-1 blocks the DNA binding cleft of TPB and the n-terminal stirrup.
<h3>Explain function of TAF(I).</h3>
- Part of a protein known as transcription factor IID can be made using instructions from the TAF1 gene (TFIID).
- The body's cells and tissues all have this protein, which connects to and binds to DNA there.
- The majority of genes' activity is controlled by transcription factor IID.
- A preinitiation complex made up of a number of initiation components must be assembled before RNA polymerase II can begin transcription of a gene from its core promoter region.
- It is believed that the TFIID transcription factor complex, which is made up of the TATA-binding protein (TBP) and TBP-associated factors (TAF(II)s), binds to the various promoters in a sequence-specific manner to initiate the assembly of this complex.
- Here, we identify and characterize a novel multiprotein complex made up of a number of TAF(II)s and other proteins but lacking either TBP or a TBP-like factor.
- In in vitro transcription experiments, this complex can take the place of TFIID on both TATA-containing and TATA-lacking promoters.
Learn more about function of TAF(I) here:
brainly.com/question/9779261
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