<span>A) Archaea and Cyanobacteria is you answer im prity sure</span>
A) s waves travels faster
A) It would die as harmful substances entered the cell
Hello friend!!
The biggest difference between the two is that the nervous system uses electrical impulses to send signals through neurons, whereas the hormonal system uses chemical messengers transported into blood plasma to target cells.
Transmission by the nervous system is short-lived but quick, whereas transmission by the hormonal system is long-lasting but takes much longer. Hene, communication is faster when using the nervous system.
In the nervous system, responses are localised - whereas in the hormonal system, they are widespread. Responses are often permanent in the hormonal system, but temporary and reversible in the nervous system.
Hope It Helps!!All The Best!!
Answer:
1. Acetylcholine binds to receptors on the motor end plate
2. Ligand-gated channels open leading to depolarization
3. End plate potential triggers an action potential
4. Transverse tubules convey action potentials into the interior of the muscle fiber
5. Calcium is released from the sarcoplasmic reticulum
6. Calcium ions bind to troponin, which then moves tropomyosin
Explanation:
Acetylcholine (ACh) is a signaling molecule (neurotransmitter) that binds to receptors on muscle cells. This binding triggers the opening of ligand-gated sodium channels, thereby ions enter into muscle cells, which causes the depolarization of the sarcolemma and thus promotes the release of Ca2+ ions from the sarcoplasmic reticulum. The myoneural junction, also known as the motor endplate, is the site of synaptic contact between a motor axon and a skeletal muscle fiber. The endplate potential is the voltage that produces the depolarization of muscle fibers when ACh molecules bind to their receptors in the cell membrane. This depolarization spreads in the sarcolemma through transverse tubules (T tubules) and thus generates an action potential. Finally, this action potential induces the release of Ca2+ in the sarcoplasmic reticulum, which activates troponin protein and induces muscle contraction.
