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A case study of the effects of mutation: Sickle cell anemia. Sickle cell anemia is a genetic disease with severe symptoms, including pain and anemia. The disease is caused by a mutated version of the gene that helps make hemoglobin — a protein that carries oxygen in red blood cells.
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In mendelian inheritance, the alleles for a gene show normal dominant-recessive relationship. Chromosomes also show crossover due to which new random combination of traits is possible in the offspring. This crossover takes place between homologous chromosomes during meiosis I.
Organellar DNA like the ones present in mitochondria and chloroplast do not follow mendelian inheritance because unlike nuclear chromosomes they do not have cross over events. There is no orderly segregation of alleles during meiosis. Traits controlled by them are usually inherited as it is and usually it is from the maternal parent because paternal gamete like sperm does not contain mitochondria.
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