Answer:
As, this individual is expending fructose syrup.
Fructose is changed over to purine which is additionally changed over to uric acid.
Along these lines, more fructose in the eating routine then more purines in the body which will additionally frame progressively uric acid.
This uric acid gathers and causes gout.
What is the best clarification for the introduction of gout right now please clarify why different decisions are not reliable with the information or what extra data you may need to choose the underlying driver?
1. overproduction of purines-They are not being created in the body however yes they are certainly are delivered from the abundance fructose expended.
2. diminished rescue of purines-No, this isn't the main driver.
3. diminished urinary discharge of uric acid this can occur as less uric acid is being discharged in the pee.
When, contrasted with the ordinary individual the patient is discharging less uric acid.
Along these lines, third point is the purpose behind gathering of uric acid in the body.
Answer:
of the epidermis
Explanation:
It forms the outermost layer of the skin
protects the body against various external influences, such as mechanical stress, radiation, microbial and penetration.
Answer:
<u>Ethanol prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u>
Explanation:
Ethylene glycol is an organic compound usually used in antifreeze solutions, solvents and cleaners.
It is abused during self-destruction and accidental intakes.
<u>In the body, ethylene glycol is acted upon by alcohol dehydrogenase and is converted into glycolate and oxalate.</u>
Glycolate and oxalate are both nephrotoxic/ kidney damaging substances. Oxalate precipitates calcium oxalate stones in the kidney. Ethylene glycol poisoning also causes high anion gap metabolic acidosis.
In order to prevent ethylene glycol poisoning, the patient is infused with ethanol, ethanol <u>prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u> In this way, ethylene glycol is not metabolized and the formation of nephrotoxic substances is prevented. Alcohol dehydrogenase instead reacts with ethanol to form acetaldehyde.
can u pls explain the question a bit more clearly
