<span> In order for the living of the species, when they reproduce, they do so in the millions so that a some of the progeny will be bound to survive.</span>
Answer:
No, there are multiple ways in which different mutations in the same gene can cause the same phenotype
Explanation:
Several different mechanisms of mutation can lead to the same phenotype. For example, lets say our phenotype is that flies have white eyes, and we know that this occurs in one particular gene that normally makes the eye colour red. (the red gene)
These mutations likely rendered the red gene ineffective (as the eyes are not red). However, this could happen in a variety of ways.
- There could be a single base deletion in the first exon of the mRNA, changing the reading frame of the protein and messing up the entire sequence (a frame shift mutations)
- The entire gene could be deleted
- A single base could be substituted in an important site of the gene, for example, one which translates into a catalytic residue or binding site in the protein
- There could be an inversion at the promoter region of the gene, such that a transcription factor can no longer bind to transcribe the gene.
There are countless other ways in which a mutation could have been caused. Therefore, just because we know the same gene is affected does not mean that we can assume the mutations are identical.
Deforestation is not a natural change.
Answer:
Zika virus and West Nile virus are the two pathogens which causes more diseases due to increase the population of mosquito.
Explanation:
Zika virus belongs to the family of Flaviviridae. Aedes mosquitoes which are active at day time are the carrier of this virus. West Nile virus is also spread through the bite of mosquito. The infected mosquito is the main cause of spreading of this disease. This virus is spreading too fast in the United States of America in the summer season where the population of mosquitoes increases.
Explanation:
The use of CRISPR/Cas9 avoids the need for protein engineering to develop a site-specific nuclease against a specific DNA target sequence, requiring only the synthesis of a new piece of RNA. This dramatically simplifies and greatly reduces the time needed for gene editing design and implementation.
