Answer:
Explanation:
They can adapte to fresh water but they can also look for the salt i think
Answer:
Since there is no distinction in the measure of the RNA polymerases yet rather their movement, the distinction lies in their structure and not their grouping. Adjustments are made to widen the states of endurance. Thus E. coli would not constrain it's endurance by restricting its development to hotter temperatures. Thus the appropriate response is "the RNA polymerase sub-units of the P. syringe strain most likely have additional adaptability with the goal that they can move all the more openly in colder temperatures".
When the rate of repair lags behind the rate of mutation, the possible fate of the cell is that (A) The cell can be transformed into a cancerous cell.
Mutations are the changes in the sequence of genes. If the rate of repair lags behind, there is a major possibility that the cell can turn into a cancerous one. This is because mutations are a big reason for the appearance of cancer.
A cancerous cell is the one that proliferates at an excessive rate. The most important property of cancerous cell is of metastasis. It is the movement of the tumor into different parts of the body and infect the healthy cells of that area as well.
To know more about cancerous cells, here
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Answer:
Explanation:
NADH and FADH2 are both electron carriers of the electron transport chain. NADH gives up its electrons starting from Complex I, which has a higher energy level compared to other complexes. Energy is given off to pump protons across the membrane by the time electrons are transferred to ComplexIII. More electrons are pumped across the membrane as electrons move to Complex IV. Because NADH commenced giving up its electrons from Complex I (higher energy level complex), more protons are pumped across the membrane gradient, which enables ATP synthase with more power to produce 3ATP molecules per NADH molecule.
On the other hand, 2 molecules of ATP are generated by FADH2 because it starts by giving up its electrons to ComplexII. It missed a chance to pump protons across the membrane when it passed Complex I. By the time the electrons reach Complex IV, less protons have been pumped. The lesser the protons to power ATP synthase, the lesser the ATP molecules produced.
