Answer:
Tight junctions - prevent liquid from seeping between cells
Desmosomes - act as rivets to hold adjacent cells together when epithelial tissue moves
Gap junctions - allow movement of cytosol, ions, and small molecules between animal cells
Plasmodesmata - openings through the cell walls of plant cells that allow adjacent cells to share materials
Explanation:
1) Tight junctions are one of the cell junctions found in animal cells. Tight junctions function to prevent the flow of liquid materials between cells.
2) Desmosomes are another type of cell junctions whose function is to form a connection between two adjacent cells. The structure formed by this connection confers strength upon the tissues involved.
3) Gap junctions are the most commonly found cell junctions found in animal cells that connects adjacent cells allowing the passage of cytosol, ions and other small molecules in them from one adjacent cell to another.
4) Plasmodesmata is a cell junction found in plant cells. They are small openings lying across the cell wall of plant cells whose function is to connect the cells and facilitate the movement of materials from one cell to another.
hi there
tour answer to this is : Adenosine Triphosphate)
A high-energy molecule that cells use for processes such as growth and movement.releases energy from glucose
i hope this helps out
have a great morning
FaithRawlins14
Answer:
The correct answer is 3: "<em>High levels of Ca2+ are expected to be found </em><em>within the sarcoplasmic reticulum</em>".
Explanation:
Muscular contraction is a highly regulated process that depends on free calcium concentration in the cytoplasm. Amounts of cytoplasmic calcium are regulated by <u>sarcoplasmic reticulum</u> that functions as a storage of the ion.
When a nerve impulse reaches the membrane of a muscle fiber, through acetylcholine release, the membrane depolarizes producing the entrance of calcium from <u>extracellular space</u>. The impulse is transmitted along the membrane to the sarcoplasmic reticulum, from where calcium is released. At this point, <em>tropomyosin is obstructing binding sites for myosin on the thin filament</em>. The calcium channel in the sarcoplasmic reticulum controls the ion release, that activates and regulates muscle contraction, by increasing its cytoplasmic levels. When <em>calcium binds to the troponin C</em>, <em>the troponin T alters the tropomyosin by moving it and then unblocks the binding sites,</em> making possible the formation of <em>cross-bridges between actin and myosin filaments.</em> When myosin binds to the uncovered actin-binding sites, ATP is transformed into ADP and inorganic phosphate.
Z-bands are then pulled toward each other, thus shortening the sarcomere and the I-band, and producing muscle fiber contraction.
