Hi there!
Your answer would be
C. Observation, hypothesis, experiment, and conclusion.
Hope this helped and please give brainliest!!
Answer:
B. diffusion
Explanation:
Because diffusion is a passive process, it does not require energy expenditure by the cell.
<span>The trick here is to understand the definition of each of the cellular transport or function mechanisms listed. These are some interesting (and strange) analogies!
Facilitated Diffusion
This is when a mechanism assists in diffusing (spreading) some material into an environment. The dog on the wagon going through a spring loaded door would shoot it out into the environment. This is an odd analogy but Point 3 would be the one.
Active Transport
Is when energy is expended to transport molecules somewhere against a concentration gradient or some other barrier. Examples include transporting molecules across a cell wall. The best analogy is the dog being dragged into a bathtub (Point 1).
Phagocytosis
This is when a larger cell consumes a molecule often like eating. This matches to point 2 - the child eating the doughnut.
Passive Diffusion
Is when a concentration of molecules naturally diffuse into an environment. This suits point 5 - the crowded room full of people.
Pinocytosis
Is the budding of cell membranes to consume liquid in the surrounding environment. I guess a woman drinking tea is the closest analogy listed (Point 4).</span>
The correct answer is that mutant cells will exhibit diminished oxygen consumption; decreased glycolysis results in decreased Kreb's cycle and electron transport chain.
The PFK2 enzyme catalyzes the generation of F26BP, this binds with the allosteric site of PFK-1 and increases the affinity of PFK-1 with F6P and also decreases the affinity of allosteric inhibitors citrate and ATP to PFK-1. Thus, PFK-1 will combine with F6P at a greater rate.
This ultimately results in more glycolysis, thus, more ETC and more consumption of O2. If there is no PFK2, then there will be a reduction in glycolysis, TCA, ETC, and consumption of oxygen.
The PFK2 is an enzyme accountable for monitoring the rates of gluconeogenesis and glycolysis in the human body. In the absence of glycolysis, there will be a reduction in TCA, ETC, and consumption of O2.