<h2>Exhaustion stage of general adaptation.</h2>
Explanation:
The final stage of general adaptation theory is known as exhaustion stage.
The general adaptation is has been divided into three stages- Alarm stage, resistance stage and exhaustion stage.
The theory of adaptation describes the physiological changes that an organism undergoes when it is exposed to stress.
The first stage,which is the alarm stage which is characterized by the burst of energy and the stressor acts as a stimulus.
The second stage is the resistance stage when the organism develops the resistance against stressor.
The last stage is the exhaustion stage when the energy is sufficiently depleted and the final adjustment is attained.
Answer:
photosynthesis is a process utilized by autotrophs to produce the products required for cellular respiration. Unlike you and me, plants and certain other Autotrophs like bacteria; can’t consume other organisms. That being said, they still require nutrients to preform cellular respiration. Those nutrients are obtained through processes such as photosynthesis.
So in other words photosynthesis produces glucose and oxygen required for aerobic/anaerobic cellular respiration, and that’s how there related.
Not sure my dude. My guess would have to be A
Answer:
In the genetic code, a stop codon (or termination codon) is a nucleotide triplet within messenger RNA that signals a termination of translation into protein
Explanation:
Proteins are based on polypeptides, which are unique sequences of amino acids. Most codons in messenger RNA (from DNA) correspond to the addition of an amino acid to a growing polypeptide chain, which may ultimately become a protein. Stop codons signal the termination of this process by binding release factors, which cause the ribosomal subunits to disassociate, releasing the amino acid chain. While start codons need nearby sequences or initiation factors to start translation, a stop codon alone is sufficient to initiate termination.
X-linked disorder is a recessive autosomal disorder, which means that
There is a 1 in 4 chance (25%) of having a child who is unaffected.
There is a 1 in 4 chance (25%) of having a child who is affected by the condition. These children have two copies of the gene that do not work correctly.
There is a 1 in 2 chance (50%) of having a child who is a carrier of the condition. These children have one working copy of the gene and one copy of the gene that does not work correctly.
