Answer:
Refined Vegetable and Seed Oils. Refined vegetable- and seed oils include corn, sunflower, safflower, soybean and cottonseed oils. ...
BPA. ...
Trans Fats. ...
Polycyclic Aromatic Hydrocarbons (PAHs) ...
Coumarin in Cassia Cinnamon. ...
Added Sugar. ...
Mercury in Fish.
Answer:
Kerb cycle
<u>Explanation</u>:
The result from mitochondria is ATP, that acts as a currency of the energy in the cell. In the cytoplasm of mitochondria kerb cycle occurs. In this process it has several other process, once it uses acetate and water to give NADH from NAD+ and gives carbon dioxide as waste. The NADH formed is fed by oxidative phosphorylation or electron transport. The outcome of these processes is oxidation of nutrients to give chemical energy in ATP form.
The correct answer is "the formation of new brain cell connections as the result of reading a book".
Usually, neuroplasticity is apparent in young children when the central nervous system has an abundance of neurons. This is always in the context of forming new brain cell connections or synapses that will lead the person to access the information quicker. This also exemplifies the principle of use and disuse when it comes to knowledge.
The other choices concerns the skeletal system, cardiovascular system, and the endocrine/reproductive system; all of which do not have a relation with neuroplasticity.
C. DNA codes for RNA which codes for proteins
<span>Rhabdomyolysis constitutes a common cause of acute renal failure and presents paramount interest. A large variety of causes with different pathogenetic mechanisms can involve skeletal muscles resulting in rhabdomyolysis with or without acute renal failure. Crush syndrome, one of the most common causes of rhabdomyolysis presents increased clinical interest, particularly in areas often involved by earthquakes, such as Greece and Turkey. Drug abusers are another sensitive group of young patients prone to rhabdomyolysis, which attracts the clinical interest of a variety of medical specialties.
We herein review the evidence extracted from updated literature concerning the data related to pathogenetic mechanisms and pathophysiology as well as the management of this interesting syndrome.
Keywords: Rhabdomyolysis, acute renal failure, myoglobin, crush syndrome
The first case of the crush syndrome, which constitutes one of the main causes of rhabdomyolysis, was reported in Sicily in 1908, after an earthquake1,2. In 1930, in the Baltic area, an epidemic of myoglobinuria was observed due to consumption of contaminated fish. Interest in rhabdomyolysis and crash syndrome was stimulated during the World War II particularly after the bombing in London, where the victims developed acute renal failure and myoglobinuria1.
Rhabdomyolysis is a rupture (lysis) of skeletal muscles due to drugs, toxins, inherited disorders, infections, trauma and compression3. Lysis of muscle cells releases toxic intracellular components in the systemic circulation which leads to electrolyte disturbances, hypovolemia, metabolic acidocis, coagulation defects and acute renal failure due to myoglobin4.
The skeletal muscle consists of cylindrical myofibrils, which contain variant structural and contraction proteins. Actin and myosin, arranged in thin and thick filaments respectively, form the repeated functional units of contraction, the sarcomeres5. The sarcoplasmic reticulum constitutes an important cellular calcium storage. It is structurally connected to the t-tubules, that are formed by invaginations of the muscle cell plasma membrane, the sarcelemma, around every fibril (Figure 1). After the sarcelemma depolarization, the stimulation arrives, through the t-tubules junctions, at the sarcoplasmic reticulum, inducing the calcium ions release and triggering muscle contraction6.</span>
