Answer:
Elevated uric acid levels lead to a condition known as "Hyperuricemia."
Explanation:
This condition occurs when there is an excessive presence of uric acid in the blood. It can be divided into:
- Primary hyperuricemia
- Secondary hyperuricemia
Primary hyperuricemia occurs when the kidneys are not able to eliminate uric acid in an efficient/proper way. This can be due to the consumption of foos that are very high in purines. For example, liver, gravy, alcoholic beverages, and some vegetables such as spinach and asparagus.
Secondary hyperuricemia may occur to several possible reasons. For instance, kidney disease, in which the kidney is not able to eliminate uric acid; chemotherapy, which may lead to the accumulation of uric acids; or medications that increase the level of uric acid in the blood.
Symptoms include:
- Fever.
- Extreme and abnormal fatigue.
- Joint inflammation.
- Urination problems.
- Kidney stones.
To prevent this disease and as part of a treatment (along with medications prescribed by a doctor), a patient should opt to consume foods that are low in purine, such as lettuce, tomatoes, cereals, fruits, milk, eggs, amongst others.
Answer: DNA is a long polymer with deoxyriboses and phosphate backbone. Having four different nitrogenous bases: adenine, guanine, cytosine and thymine. RNA is a polymer with a ribose and phosphate backbone. Four different nitrogenous bases: adenine, guanine, cytosine, and uracil.
Explanation:
Answer:
Adenine and guanine are purines, while thymine, cytosine and uracil are pyrimidines.
Hope this helps! Sorry no one answered your question sooner :(
Answer: The Calvin cycle has four main steps: carbon fixation, reduction phase, carbohydrate formation, and regeneration phase. Energy to fuel chemical reactions in this sugar-generating process is provided by ATP and NADPH, chemical compounds which contain the energy plants have captured from sunlight.
Explanation:
Answer:
a. resolve the branching patterns (evolutionary history) of the Lophotrochozoa
b. (the same, it is repeated)
Explanation:
Nemertios (ribbon worms) and foronids (horseshoe worms) are closely related groups of lofotrocozoa. Lofotrocozoans, or simply trocozoans (= tribomastic celomados with trocophoric larva) are a group of animals that includes annelids, molluscs, endoprocts, brachiopods and other invertebrates. They represent a crucial superphylum for our understanding of the evolution of bilateral symmetry animals. However, given the inconsistency between molecular and morphological data for these groups, their origins were not entirely clear. In the work linked above, the first records of genomes of the Nemertine worm Notospermus geniculatus and the foronid Phoronis australis are presented, along with transcriptomes along the adult bodies. Our phylogenetic analyzes based on the genome place Nemertinos as the sister group of the taxon that contains Phoronidea and Brachiopoda. It is shown that lofotrocozoans share many families of genes with deuterotomes, suggesting that these two groups retain a common genetic repertoire of bilaterals that do not possess ecdisozoans (arthropods, nematodes) or platizoos (platelets, sydermats). Comparative transcriptomics demonstrates that foronid and brachiopod lofophores are similar not only morphologically, but also at the molecular level. Although the lofophore and vertebrates show very different cephalic structures, the lofophorees express the vertebrate head genes and neuronal marker genes. This finding suggests a common origin of the bilaterial pattern of the head, although different types of head will evolve independently in each lineage. In addition, we recorded innate immunity expansions of lineage-specific and toxin-related genes in both lofotrocozoa and deuterostomes. Together, this study reveals a dual nature of lofotrocozoans, in which the conserved and specific characteristics of the lineage shape their evolution.
