Answer:
C. +23.1 kJ/mol
Explanation:
the formula to use to calculate the energy requirement in kJ/mol to transport a proton across the mitochondrial inner membrane in plant cells is:
ΔGt = RTIn 
+ ZFΔV
let's list the values of the data we are being given in the question to make it easier when solving it.
Z= 1
F= 96500C (faraday's constant)
ΔV= 160mV = 0.160V
R= 8.314( constant)
T= 15ºC ( converting our degree Celsius into kelvin, we will have 273.15k+ 15 = 288.15K)
∴ T= 288.15K
Putting it all together in the formula, we have:
ΔGt = 8.314 × 288.15 × 2.303 log + 1 × 96500 × 0.160
ΔGt = 5517.25 
+15440
ΔGt = 5517.25 
+15440
Given that the pH differential gradient across the membrane is 1.4pH units. It implies that;
ΔGt = 5517.25 × 1.4 + 15440
= 7724.15 +15440
= 23164.15 Joules/moles
= +23.1 KJ/mole
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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Many fruits contain specialized structures that are adaptations for The answer is D. aiding in seed dispersal.
Maple trees have fruits with "Wings" that help the gale disperse the seeds. Some flowering plants grow fleshy fruit that benefits disperse their seeds. When animals eat the fruit, the seeds pass through an animal's digestive tract undamaged.
Answer:
Cancer can develop anywhere in the body. It starts when cells grow out of control and crowd out normal cells. This makes it hard for your body to work the way it should.
For many people, cancer can be treated successfully. In fact, more people than ever before lead full lives after cancer treatment. The cells in our bodies all have certain jobs to do. Normal cells divide in an orderly way. They die when they are worn out or damaged, and new cells take their place. In cancer, the cells keep on growing and making new cells. They crowd out normal cells. This causes problems in the part of the body where the cancer started. They can also spread to other parts of the body.
B. How far do butterflies migrate
A, C and D are opinions not facts.
