True
because ur hart pumps faster and then blood flow increases and the you have more blood delivery to your body
<h2>GPCRs and RTKs</h2>
Explanation:
1) Phosphorylation of receptor can terminate signal or desensitize receptor: GPCRs because when signal persists a protein kinase receptor is activated,this further catalyse the phosphorylation of several serine residue at cytosolic phase of 7 transmembrane
Phosphorylated transmembrane recognized by arrestin protein blocks interaction between trimeric G protein and receptor thus blocks signal transduction
2) Autophosphorylation of receptor can initiate signal: RTKs because receptor dimerization activates intrinsic tyrosine kinase activity by which they phosphorylate its own tyrosine residue on cytosolic phase
3) Structure contains seven transmembrane helices: GPCRs because it consists of single polypeptide and spans the membrane 7 times hence called 7 transmembrane;it recognizes external signal and activates trimeric G protein
4) Ligand binding induces conformational change in receptor: RTKs because normally RTK exist in monomeric form in absence of any ligand molecule but when any ligand molecule binds receptor starts to dimerize
5) Receptor activation causes phosphorylation of its cytosolic subunits: RTKs because when receptor gets activates the intrinsic tyrosine kinase activity by which they phosphorylate its cytosolic subunits
6) An example is the insulin receptor: Insulin receptor is present on plasma membrane and belongs to RTK family and always exist in dimeric form
7) Transports some ligands through the membrane: Neither GPCR nor RTK;ligand gets transported through the membrane through ligand gated channel
8) An example is the epinephrine receptor: Epinephrine is an endocrine hormone produced by adrenal glands in stress condition and prepare body for fight and flight;Epinephrine uses GPCR signalling pathway by four different types of membrane receptors in different tissues
9) Activate heterotrimeric G proteins directly: Transmembrane protein of GPCR recognizes the external signal and activates G protein,activated G protein binds with effector enzyme and activate it which further produce or destroy secondary messenger that carry message from cell surface to cell interior
Each myofibril is attached at either end of the muscle fiber, when sarcomeres shorten, the muscle fiber Shortens
Mechanism :
Myofibrils are components of the animal skeletal muscles that are made up of sacromeres. They perform the muscular contraction and relaxation via the sliding filament model.
To produce motion, A cross-bridge forms between actin and the myosin heads triggering shortening of the sacromere. This shortening of the sacromere results in the contraction of the muscle fibers, which the net effect can be seen to be a shortening of the fibers.
To learn more about myofibril refer to : brainly.com/question/15461128
#SPJ4
C. When exposed to antibiotics, most bacteria in a population die but some survive and live to reproduce.
The correct answer is: D. The red blood cells were placed in a hypotonic solution. Water moved from the outside of the cells to the inside of the cells until the cells burst.
Hypotonic solution can be described as solution that has less solute and more water (H2O) than another solution. In order to maintain homeostasis, the water will move (via osmosis) into the cell, through the semipermeable membrane of the red blood cell.
