Answer:
It is the cardiac output.
Explanation:
Heart rate x stroke volume = cardiac output.
The cardiac output is the final outcome of all factors that influentiate the ventricular activity (heart rate, heart contractility, and the phenomenon of precharge and postcharge).
Answer:
D
Explanation:
Quizlet is amazing for answers <3
Answer:
Correct answer is c. It is the final electron acceptor in the aerobic respiration.
Explanation:
Oxygen is a substrate of the aerobic respiration, but it is not the only one. Glucose is also a substrate.
Oxygen is used in the cells to be the final electron acceptor, this means that receives the electrons from NADH and FADH2. That is why, when there is no oxygen available for aerobic respiration, the NADH and FADH2 cannot be oxidized and therefore remain in their reduced form. As a consequence, they cannot be re-utilized during different cellular processes that are NAD+ and FAD dependant, such as glycolysis, pyruvate oxidation and cellular respiration. This means that the ATP synthesis stops.
Oxygen itself does not transport any electrones, this are transported by the cytochrome complex in the mitochondrial membrane. But oxygen is key in receiving those electrones, therefore a very important piece of the electron transport across the mitochondria.
Answer: 1
Explanation:
Subtrack 2-1 and get 1. The proof it's 1 add 1+1 and get 2
Answer and explanation :
there are many problem due to lack of pneumotaxic and stetch receptor neurons on ventilation
DUE TO LACK OF PNEUMOTAXIC CENTER :
- pneumotaxic center is present in the nucleus which is used to transmit impulse to the inspiratory area which is not possible due to lack of pneumotaxic center
- it controls the duration of filling of lungs
- it also control the medullary respiratory system which is not possible due to lack of this
DUE TO LACK OF STRETCH RECEPTOR :
- The hearing breuer inflation reflex is generated by stretch receptor neuron which is not generated due to lack of stretch receptor
- when stretched these receptors send the inhibitory impulses through the vegus nerve to the DMG neuron which is not possible due to lack of this
