The archean era i think am i right?
The correct option is B.
Rockfeller asked Diego to make an artistic work for the lobby of the RCA building. The final work which Diego presented contained the portrait of Vladimir Lehin although this was not part of the contract arrangement. Vladimir Lenin was an hated politician, who is synonymous with communism and Rockefeller did not share the political ideas of this man at all, because of this Rockefeller asked Diego to remove the artwork; the artist refused and the artwork was eventually destroyed.<span />
Answer: Claim 1: because Forests play a huge role in the carbon cycle on our planet. When forests are cut down, not only does carbon absorption cease, but also the carbon stored in the trees is released into the atmosphere as CO2 if the wood is burned or even if it is left to rot after the deforestation process.
Explanation:
The stamen is the part of the flower that produces pollen.
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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