Answer:
I would say the protein that help the skin is the underlying tissue
Explanation:
Answer:
The correct answer is B.
Explanation:
During the follicular phase (first half of female cycle), follicles in the ovary begin developing under the<u> influence of </u><u>FSH.</u> <em>The follicle that acquires more FSH receptors will become </em><em>dominant</em> and will produce more estrogen and inhibin hormone than the others. Inhibin will reduce FSH level and as a result the other follicles will fail to keep growing. At this stage the dominant follicle will become FSH independent.
Estrogen produced by the dominant follicle will stimulate LH secretion. After approximately 24-36 hours from when LH reaches its peak level, the dominant follicle releases an ovocyte. <em>This event is called ovulation.</em>
Answer:
The answer is letter c, The structure of individual neurons.
Explanation:
Brain-imaging technique- <u><em>this comprises of different techniques for analyzing the brain's activities as well as structure, function and other problems.</em></u>
There are many brain-imaging techniques such as <u>Positron Emission Tomography or PET,</u> which traces the part of the brain that is activated during specific tasks as well as the localization of brain activity in response to a specific stimulus. These areas are seen with high radioactivity.
Patterns of blood flow in the brain can be detected by using <u>MRI (Magnetic Resonance Imaging) test.</u> There is an increase in blood flow to areas in the brain with high activity level.
With the explanation given, it shows that letter c is the only choice that the brain-imaging techniques cannot determine.
Answer:
Explanation:
a. Name, phone and contact and self explanatory, it may be d, but that’s doubtful because I’d assume it means the type of doctor Eg; MD, DO, DNM, DHM, etc.
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
