Snapper overfishing
Explanation:
Coral reefs are becoming highly vulnerable due to increased anthropological exploitation of the ocean reserves. Overfishing, usage of non-selective fishing gears, fishing the spawning areas, fishing using nets etc all affect the coral reefs and the marine ecosystem. The coral reefs and the fish population are interdependent and overfishing greatly impacts both the factors.
Overfishing is the main reason for damage and decline in coral reefs. The snappers are carnivores and survive on eating other fishes which can be herbivores or omnivores. Snappers remain at the top of the food pyramid of the marine ecosystem. A reduction at the top level of the food pyramid will automatically disturb the lower level population and the energy flow between each cycle. A reduction in carnivores leads to increase in herbivores which in turn deplete the coral reefs. Coral reef is a habitat for many fish populations, and a reduction in the habitat will again reduce the overall population of fishes. A decrease in predator population will increase prey population.
There are different trophic levels at each stage. Trophic levels determine the amount of energy at each stage of the food chain. For example, the energy is 100% at the producer (plant), so it's the 1st trophic level, and so on. Energy amounts decrease as you go up a food chain, and you rarely find more than 5 animals in one because there is not enough energy left for the 6th consumer
<span>After the third month of development, lanugo develops on the head (thin, first hair), the muscles and bones are formed an shaped, the mouth movement is possible, the liver and pancreas produce fluid secretions, meconium is made (stool of infant), reproductive organs are developed, but the baby's gender is difficult to distinguish on ultrasound.</span>
Answer:
Neutrophils help fight infections because they ingest microorganisms and secrete enzymes that destroy them. A neutrophil is a type of white blood cell, a type of granulocyte and a type of phagocyte.
Explanation:
Neutrophils display adhesion glycoproteins on their surface to bind endothelial and subendothelial structures. They move randomly until they find a damaged site. Unless neutrophils are activated, endothelial cells do not tend to adhere. When inflammation mediators (IL-1, FNT) activate endothelial cells, they express P-selectin and E-selectin on the surface. The expression of glycoproteins and L-selectin cause the initial adhesion of the non-stimulated neutrophil to the activated endothelium, slowing it down by rolling it over the endothelium. Activated endothelial cells, opsonized particles, immune complexes, FEC-G, FEC-GM and chemoattractants produce factors that stimulate neutrophil activation. Expressing β2 integrin (endothelium adhesion molecule) Neutrophils expand and form pseudopods. Neutrophil activation also promotes degranulation, superoxide generation, and arachidonate metabolite production.
